
Open repository of veterinary pathology slides contributed by individuals and institutions around the world.
Thumbnail | Full_Size | IMAGE_ID | IMAGE_FRAME | Image | Contributor | Institution | Species | System | Organ | General Pathology Category | SUBSET_ID | Diagnosis | Description | Cause | IMAGE_ACTIVE | IMAGE_VISIBILITY | IMAGE_KEYWORD | Comments | VSPO Link | Related Images | WSC Link | Upload Date |
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123,610 | 64,715 | F64715 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | LIVER | TOXICOLOGIC | 32 | Marked diffuse acute hepatic swelling | Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage) | 1 | 1 | Salvia reflexa; diterpinoids; hepatocellular necrosis; 7-hydroxyrhyacephiline; jaundice | Salvia reflexa was recently identified as hepatoxic to cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The liver is from a four-year old Aberdeen Angus cow. The poisoning episode in Wyoming had many features in common with the episode in the Colorado herd described by Panter et al. A large number of cattle became anorectic, depressed, aggressive and recumbent within 12 – 24 hours of exposure to toxic hay. Toxic principals includes 7-hydroxyrhyacephiline,a novel seco-clerodane diterpene. All parts of Salvia reflexa are toxic, except the seeds, with the highest concentration in calyces. Image courtesy of Dr. Brett T. Webb, WSVL#23B1809. | 03/28/2023 | ||||
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123,611 | 64,716 | F64716 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | ELK | RESPIRATORY | LUNG | PARASITE | 32 | Parasitic bronchitis with intralesional Dictyocaulus sp. | Dictyocaulus sp. (cervi or viviparus) | 1 | 1 | Parasite; parasitic bronchitis; Dictyocaulus cervi; Dictylocaulus viviuparus; chronic wasting disease | The longstanding suspicion that not all lungworm in Wyoming’s free-ranging elk are Dictyocaulus viviparus was strengthened recently. Some have morphological features of D. cervi, a slightly longer nematode, and are a presumed import of European origin. Bangoura et al. Dictyocaulus cervi-like lungworm infection in a Rocky Mountain elk (Cervus Canadensis Nelsoni) from Wyoming, USA: 2021, J Wildl Dis 57(1), 71-81. | 03/28/2023 | ||||
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123,612 | 64,717 | F64717 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | ELK | NERVOUS | BRAIN | NORMAL | 32 | Grossly normal brain (CWD-positive) | CWD | 1 | 1 | Chronic wasting disease; emaciation | The brains of elk with CWD are grossly unremarkable. Aspiration pneumonia with emaciation, both of which were present in this elk, are the principal gross signs of clinically advanced CWD. WSVL#20W7606 | 03/28/2023 | ||||
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123,613 | 64,718 | F64718 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | ELK | HEMATOPOIETIC | BONE MARROW | ATROPHY | 32 | Serous atrophy of fat, femoral bone marrow | CWD | 1 | 1 | Chronic wasting disease; emaciation; serous atrophy of fat | Emaciation and serous atrophy of fat are the principal gross findings in terminal CWD. The image is of mid-shaft femoral bone marrow. WSVL#20W7606 | 03/28/2023 | ||||
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123,614 | 64,719 | F64719 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | ELK | BODY AS A WHOLE | BODY AS A WHOLE | ATROPHY | 32 | Emaciation | CWD | 1 | 1 | Chronic wasting disease; emaciation | Emaciation with serous atrophy of fat, both of which were present in this CWD-positive elk, are the principal gross signs of clinically advanced CWD. WSVL#20W7606 | 03/28/2023 | ||||
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123,615 | 64,720 | F64720 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | Pronghorn (Antilocapra americana) | URINARY | KIDNEY | INFECTIOUS | 32 | Pyelonephritis with septicemia | E. coli | 1 | 1 | Pyelonephritis; septicemia; E coli | WSVL#20W3089 | 03/28/2023 | ||||
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123,616 | 64,721 | F64721 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | Pronghorn (Antilocapra americana) | URINARY | KIDNEY | INFECTIOUS | 32 | Pyelonephritis with septicemia | E. coli | 1 | 1 | Pyelonephritis; septicemia; E coli | WSVL#20W3089 | 03/28/2023 | ||||
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123,617 | 64,722 | F64722 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | LIVER | TOXICOLOGIC | 32 | Acute hepatocellular necrosis | Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage) | 1 | 1 | Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis | Histological appearance of liver showing marked acute periacinar necrosis and microvacuolation of hepatocytes. Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes in lance-leaf sage.. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx. | 03/28/2023 | ||||
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123,618 | 64,723 | F64723 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | LIVER | TOXICOLOGIC | 32 | Acute hepatocellular necrosis | Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage) | 1 | 1 | Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis | Histological appearance of liver showing marked acute periacinar necrosis and early biliary hyperplasia. Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes in lance-leaf sage.. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx. | 03/28/2023 | ||||
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123,619 | 64,724 | F64724 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | LIVER | TOXICOLOGIC | 32 | Acute massive hepatocellular necrosis | Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage) | 1 | 1 | Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis | Histological image shows massive necrosis of all portions of the lobule; some autolysis with bacterial overgrowth is present. Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx. | 03/28/2023 | ||||
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123,620 | 64,725 | F64725 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | LIVER | TOXICOLOGIC | 32 | Acute massive hepatocellular necrosis | Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage) | 1 | 1 | Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis | Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx. | 03/28/2023 | ||||
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123,621 | 64,726 | F64726 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | LIVER | TOXICOLOGIC | 32 | Acute massive hepatocellular necrosis | Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage) | 1 | 1 | Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis; plant | Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx. | 03/28/2023 | ||||
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123,622 | 64,727 | F64727 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | LIVER | TOXICOLOGIC | 32 | Acute massive hepatocellular necrosis | Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage) | 1 | 1 | Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis | Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx. | 03/28/2023 | ||||
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123,594 | 64,699 | F64699 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | ABDOMINAL CAVITY | ABDOMEN | OBSTRUCTION | 32 | Abdominal distention with subcutaneous hemorrhage and edema, secondary to uroperitoneum | Urolilithiasis | 1 | 1 | Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis | WSVL#22B7593 | 03/27/2023 | ||||
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123,595 | 64,700 | F64700 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | URINARY | URETHRA | OBSTRUCTION | 32 | Necrotizing urethritis, severe, subacute | Urolilithiasis | 1 | 1 | Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis | The severe urethritis exacerbated urinary tract obstruction in this animal. WSVL#22B7593 | 03/27/2023 | ||||
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123,596 | 64,701 | F64701 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | URINARY | URINARY BLADDER | OBSTRUCTION | 32 | Ruptured bladder with hemorrhage | Urolilithiasis | 1 | 1 | Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis | In cattle, bladders tend to split along the dorsal aspect, longitudinally, as here. WSVL#22B7593 | 03/27/2023 | ||||
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123,597 | 64,702 | F64702 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | ABDOMINAL CAVITY | PERITONEAL CAVITY | OBSTRUCTION | 32 | Uroperitoneum, secondary to urolithiasis and necrotizing urethritis | Urolilithiasis | 1 | 1 | Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis | WSVL#22B7593 | 03/27/2023 | ||||
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123,598 | 64,703 | F64703 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | INTEGUMENT | BODY AS A WHOLE | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 1 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. Dog carcass as submitted. Note absence of blood on dog or any obvious wound. | 03/27/2023 | ||||
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123,599 | 64,704 | F64704 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | INTEGUMENT | SKIN | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 3 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. Radiograph showing location of pellet in carcass. | 03/27/2023 | ||||
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123,600 | 64,705 | F64705 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | INTEGUMENT | SKIN | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 4 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. It is surprisingly easy to miss the entry wound of a small projectile. A good approach is to skin the carcass and look at subcutis for hemorrhage, as you can see here. | 03/27/2023 | ||||
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123,601 | 64,706 | F64706 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | RESPIRATORY | THORACIC CAVITY | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 6- Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. BB pellet in thorax. BB pellets are less informative than larger projectiles, but are useful evidence and should be retained for any legal hearings. | 03/27/2023 | ||||
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123,602 | 64,707 | F64707 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | RESPIRATORY | THORACIC CAVITY | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 7 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. | 03/27/2023 | ||||
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123,603 | 64,708 | F64708 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | RESPIRATORY | THORAX | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 8 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Direction of projectile, through body wall is shown by a shorn bacteriology swab (plus Photoshopped arrow). Hemorrhagic entry wound in posterior lobe of right lung is also visible. | 03/27/2023 | ||||
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123,604 | 64,709 | F64709 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | RESPIRATORY | LUNG | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 9 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Entry wound in right lung | 03/27/2023 | ||||
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123,605 | 64,710 | F64710 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | RESPIRATORY | LUNG | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 10 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Medial aspect of right lung with two point of penetration by BB | 03/27/2023 | ||||
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123,606 | 64,711 | F64711 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | CARDIOVASCULAR | AORTA | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 11 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Trajectory of pellet through thoracic aorta (see next image – difficult to identify both penetrating and exit wounds. Peri-aortic hemorrhage. | 03/27/2023 | ||||
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123,607 | 64,712 | F64712 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | CARDIOVASCULAR | AORTA | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 12 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Trajectory of BB pellet through aorta, demonstrated by bacteriology swab. | 03/27/2023 | ||||
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123,608 | 64,713 | F64713 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | CARDIOVASCULAR | BLOOD | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 13 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Documenting volume of blood (1.25 L) in thorax. | 03/27/2023 | ||||
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123,609 | 64,714 | F64714 | O’TOOLE | UNIVERSITY OF WYOMING | CANINE | RESPIRATORY | AGENT-FOREIGN BODY | Forensic | 32 | Acute exsanguination following gunshot | Gunshot trauma | 1 | 1 | Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy | 14 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Image of BB pellet. Projectiles found in a forensic necropsy should be photographed (at a minimum), Ideally the round should be kept until legal hearings are complete. | 03/27/2023 | ||||
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123,584 | 64,689 | F64689 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | SENSES | EYE | NEOPLASIA, NEOPLASM, TUMOR | 32 | Undifferentiated carcinoma of conjunctival origin, most likely third eyelid | Neoplasia | 1 | 1 | Conjunctiva; eye; globe; carcinoma; third eyelid; keratitis | 12 year old Corgi cross with conjunctival mass and chemosis. WSVL#16C10041 | 03/26/2023 | ||||
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123,585 | 64,690 | F64690 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | FELINE | SKELETAL | BONE-SKULL | TRAUMA | 32 | Acute fracture, frontal bones. Acute hemorrhage, M. masseter | Acute trauma | 1 | 1 | Fracture; skull; frontal bones; squama; subdural hematoma; masseter | Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772 | 03/26/2023 | ||||
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123,586 | 64,691 | F64691 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | FELINE | SKELETAL | BONE-SKULL | TRAUMA | 32 | Acute fracture, frontal bones. Acute hemorrhage, M. masseter | Acute trauma | 1 | 1 | Fracture; skull; frontal bones; squama; subdural hematoma | Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772 | 03/26/2023 | ||||
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123,587 | 64,692 | F64692 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | FELINE | SKELETAL | BONE-SKULL | HEMORRHAGE | 32 | Acute intracranial hemorrhage | Acute trauma | 1 | 1 | Fracture; skull; frontal bones; intra-cranial hemorrhage | Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772 | 03/26/2023 | ||||
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123,588 | 64,693 | F64693 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | FELINE | SKELETAL | BONE-SKULL | HEMORRHAGE | 32 | Intra-cranial hemorrhage | Acute trauma | 1 | 1 | Fracture; skull; frontal bones; intra-cranial hemorrhage | Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772 | 03/26/2023 | ||||
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123,589 | 64,694 | F64694 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | NERVOUS | CEREBELLUM | DEGENERATION | 32 | Cerebellar abiotrophy OMIA 000175 (presumed) | Genetic | 1 | 1 | Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia | No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505. | 03/26/2023 | ||||
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123,590 | 64,695 | F64695 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | NERVOUS | CEREBELLUM | DEGENERATION | 32 | Cerebellar abiotrophy OMIA 000175 (presumed) | Genetic | 1 | 1 | Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia | No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505. | 03/26/2023 | ||||
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123,591 | 64,696 | F64696 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | NERVOUS | CEREBELLUM | DEGENERATION | 32 | Cerebellar abiotrophy OMIA 000175 (presumed) | Genetic | 1 | 1 | Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia | No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505. | 03/26/2023 | ||||
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123,592 | 64,697 | F64697 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | NERVOUS | CEREBELLUM | DEGENERATION | 32 | Cerebellar abiotrophy OMIA 000175 (presumed) | Genetic | 1 | 1 | Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia | No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505. | 03/26/2023 | ||||
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123,593 | 64,698 | F64698 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | NERVOUS | CEREBELLUM | DEGENERATION | 32 | Cerebellar abiotrophy OMIA 000175 (presumed) | Genetic | 1 | 1 | Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia | No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505. | 03/26/2023 | ||||
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123,575 | 64,680 | F64680 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,576 | 64,681 | F64681 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,577 | 64,682 | F64682 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,578 | 64,683 | F64683 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE-SKULL | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate, with foreshortened skull | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,579 | 64,684 | F64684 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE-SKULL | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate, with foreshortened skull and mild inferior prognathia | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,580 | 64,685 | F64685 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE-SKULL | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate, with foreshortened skull and mild inferior prognathia | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,581 | 64,686 | F64686 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,582 | 64,687 | F64687 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | SKELETAL | BONE-HUMERUS | DYSPLASIA, dysplastic | 32 | Chondrodysplasia, disproportionate | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,583 | 64,688 | F64688 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | CARDIOVASCULAR | HEART | HYPERTROPHY | 32 | Cardiac hypertrophy and chronic passive congestion | Manganese deficiency | 1 | 1 | “Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD) | This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43. | 03/25/2023 | ||||
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123,569 | 64,674 | F64674 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | CANINE | RESPIRATORY | LARYNX | FOREIGN BODY | 32 | Foreign body aspiration (food) | Aspiration of food | 1 | 1 | Foreign body; aspiration; laryngospasm | This was a nursing puppy transitioning to solid feed. Feed particles were predominantly in larynx, rather than lung. I assume the puppy died due to a combination of laryngospasm and partial obstruction by feed particles of larynx | 03/24/2023 | ||||
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123,570 | 64,675 | F64675 | O’TOOLE | WYOMING STATE VETERINARY LABORATORY | BOVINE | GASTROINTESTINAL | CECUM | METABOLIC DISEASE | 32 | Multifocal superficial ulcerative typhlitis secondary to uremia | Uremia (urolithiasis) resulting in a ruptured bladder and uroperitoneum | 1 | 1 | Uremia; cecum; typhlitis; vasculitis; urolithiasis | Cattle and horses are prone to ulcerative or hemorrhagic typhlitis-colitis as a result of uremia. By contrast, oral, lingual and gastric mucosa are the main tissues developing ulcers or hemorrhagic lesions in uremic dogs, associated with vascular lesions. WSVL#16B6800 | 03/24/2023 | ||||
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