Noah’s Arkive – 50 most recent images – The Davis-Thompson Foundation

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IMAGE_ID	IMAGE_FRAME	Image	Contributor	Institution	Species	System	Organ	General Pathology Category	SUBSET_ID	Diagnosis	Cause	IMAGE_ACTIVE	IMAGE_VISIBILITY	IMAGE_KEYWORD	Comments	Upload Date
123,610	64,715	F64715	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	LIVER	TOXICOLOGIC	32	Marked diffuse acute hepatic swelling	Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage)	1	1	Salvia reflexa; diterpinoids; hepatocellular necrosis; 7-hydroxyrhyacephiline; jaundice	Salvia reflexa was recently identified as hepatoxic to cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The liver is from a four-year old Aberdeen Angus cow. The poisoning episode in Wyoming had many features in common with the episode in the Colorado herd described by Panter et al. A large number of cattle became anorectic, depressed, aggressive and recumbent within 12 – 24 hours of exposure to toxic hay. Toxic principals includes 7-hydroxyrhyacephiline,a novel seco-clerodane diterpene. All parts of Salvia reflexa are toxic, except the seeds, with the highest concentration in calyces. Image courtesy of Dr. Brett T. Webb, WSVL#23B1809.	03/28/2023
123,611	64,716	F64716	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	ELK	RESPIRATORY	LUNG	PARASITE	32	Parasitic bronchitis with intralesional Dictyocaulus sp.	Dictyocaulus sp. (cervi or viviparus)	1	1	Parasite; parasitic bronchitis; Dictyocaulus cervi; Dictylocaulus viviuparus; chronic wasting disease	The longstanding suspicion that not all lungworm in Wyoming’s free-ranging elk are Dictyocaulus viviparus was strengthened recently. Some have morphological features of D. cervi, a slightly longer nematode, and are a presumed import of European origin. Bangoura et al. Dictyocaulus cervi-like lungworm infection in a Rocky Mountain elk (Cervus Canadensis Nelsoni) from Wyoming, USA: 2021, J Wildl Dis 57(1), 71-81.	03/28/2023
123,612	64,717	F64717	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	ELK	NERVOUS	BRAIN	NORMAL	32	Grossly normal brain (CWD-positive)	CWD	1	1	Chronic wasting disease; emaciation	The brains of elk with CWD are grossly unremarkable. Aspiration pneumonia with emaciation, both of which were present in this elk, are the principal gross signs of clinically advanced CWD. WSVL#20W7606	03/28/2023
123,613	64,718	F64718	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	ELK	HEMATOPOIETIC	BONE MARROW	ATROPHY	32	Serous atrophy of fat, femoral bone marrow	CWD	1	1	Chronic wasting disease; emaciation; serous atrophy of fat	Emaciation and serous atrophy of fat are the principal gross findings in terminal CWD. The image is of mid-shaft femoral bone marrow. WSVL#20W7606	03/28/2023
123,614	64,719	F64719	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	ELK	BODY AS A WHOLE	BODY AS A WHOLE	ATROPHY	32	Emaciation	CWD	1	1	Chronic wasting disease; emaciation	Emaciation with serous atrophy of fat, both of which were present in this CWD-positive elk, are the principal gross signs of clinically advanced CWD. WSVL#20W7606	03/28/2023
123,615	64,720	F64720	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	Pronghorn (Antilocapra americana)	URINARY	KIDNEY	INFECTIOUS	32	Pyelonephritis with septicemia	E. coli	1	1	Pyelonephritis; septicemia; E coli	WSVL#20W3089	03/28/2023
123,616	64,721	F64721	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	Pronghorn (Antilocapra americana)	URINARY	KIDNEY	INFECTIOUS	32	Pyelonephritis with septicemia	E. coli	1	1	Pyelonephritis; septicemia; E coli	WSVL#20W3089	03/28/2023
123,617	64,722	F64722	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	LIVER	TOXICOLOGIC	32	Acute hepatocellular necrosis	Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage)	1	1	Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis	Histological appearance of liver showing marked acute periacinar necrosis and microvacuolation of hepatocytes. Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes in lance-leaf sage.. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx.	03/28/2023
123,618	64,723	F64723	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	LIVER	TOXICOLOGIC	32	Acute hepatocellular necrosis	Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage)	1	1	Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis	Histological appearance of liver showing marked acute periacinar necrosis and early biliary hyperplasia. Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes in lance-leaf sage.. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx.	03/28/2023
123,619	64,724	F64724	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	LIVER	TOXICOLOGIC	32	Acute massive hepatocellular necrosis	Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage)	1	1	Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis	Histological image shows massive necrosis of all portions of the lobule; some autolysis with bacterial overgrowth is present. Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx.	03/28/2023
123,620	64,725	F64725	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	LIVER	TOXICOLOGIC	32	Acute massive hepatocellular necrosis	Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage)	1	1	Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis	Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx.	03/28/2023
123,621	64,726	F64726	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	LIVER	TOXICOLOGIC	32	Acute massive hepatocellular necrosis	Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage)	1	1	Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis; plant	Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx.	03/28/2023
123,622	64,727	F64727	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	LIVER	TOXICOLOGIC	32	Acute massive hepatocellular necrosis	Salvia reflexa (lance-leaf sage; aka wild mint, Rocky Mountain sage)	1	1	Salvia reflexa; diterpinoids; hepatocellular necrosis; jaundice; salviarin; periacinar necrosis	Salvia reflexa was recently confirmed to be hepatoxic for cattle, goats and mice (Panter KE et al: 2021, Clinical, pathologic, and toxicologic characterization of Salvia reflexa (lance-leaf sage) poisoning in cattle fed contaminated hay. J Vet Diagn Invest. 33(3):538-547). The original episode killed 165 cattle in a Colorado herd of approx. 500 (33% death loss). A more recent poisoning episode in Wyoming had many features in common with the one documented by Panter et al. Cattle become anorectic, depressed, aggressive, jaundiced and recumbent within 12 – 24 hours of exposure to toxic hay. There are at least four hepatotoxic diterpenes. The most potent is helpfully named salviarin. It occurs in all parts of the plant, except in seeds, with the highest concentration in calyx.	03/28/2023
123,594	64,699	F64699	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	ABDOMINAL CAVITY	ABDOMEN	OBSTRUCTION	32	Abdominal distention with subcutaneous hemorrhage and edema, secondary to uroperitoneum	Urolilithiasis	1	1	Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis	WSVL#22B7593	03/27/2023
123,595	64,700	F64700	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	URINARY	URETHRA	OBSTRUCTION	32	Necrotizing urethritis, severe, subacute	Urolilithiasis	1	1	Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis	The severe urethritis exacerbated urinary tract obstruction in this animal. WSVL#22B7593	03/27/2023
123,596	64,701	F64701	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	URINARY	URINARY BLADDER	OBSTRUCTION	32	Ruptured bladder with hemorrhage	Urolilithiasis	1	1	Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis	In cattle, bladders tend to split along the dorsal aspect, longitudinally, as here. WSVL#22B7593	03/27/2023
123,597	64,702	F64702	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	ABDOMINAL CAVITY	PERITONEAL CAVITY	OBSTRUCTION	32	Uroperitoneum, secondary to urolithiasis and necrotizing urethritis	Urolilithiasis	1	1	Uroperitoneum; uremia; urolithiasis; ruptured bladder; urethritis	WSVL#22B7593	03/27/2023
123,598	64,703	F64703	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	INTEGUMENT	BODY AS A WHOLE	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	1 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. Dog carcass as submitted. Note absence of blood on dog or any obvious wound.	03/27/2023
123,599	64,704	F64704	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	INTEGUMENT	SKIN	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	3 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. Radiograph showing location of pellet in carcass.	03/27/2023
123,600	64,705	F64705	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	INTEGUMENT	SKIN	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	4 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. It is surprisingly easy to miss the entry wound of a small projectile. A good approach is to skin the carcass and look at subcutis for hemorrhage, as you can see here.	03/27/2023
123,601	64,706	F64706	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	RESPIRATORY	THORACIC CAVITY	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	6- Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed hound. Malicious shooting by neighbor irritated by trespassing dog. BB pellet in thorax. BB pellets are less informative than larger projectiles, but are useful evidence and should be retained for any legal hearings.	03/27/2023
123,602	64,707	F64707	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	RESPIRATORY	THORACIC CAVITY	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	7 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog.	03/27/2023
123,603	64,708	F64708	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	RESPIRATORY	THORAX	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	8 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Direction of projectile, through body wall is shown by a shorn bacteriology swab (plus Photoshopped arrow). Hemorrhagic entry wound in posterior lobe of right lung is also visible.	03/27/2023
123,604	64,709	F64709	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	RESPIRATORY	LUNG	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	9 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Entry wound in right lung	03/27/2023
123,605	64,710	F64710	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	RESPIRATORY	LUNG	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	10 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Medial aspect of right lung with two point of penetration by BB	03/27/2023
123,606	64,711	F64711	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	CARDIOVASCULAR	AORTA	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	11 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Trajectory of pellet through thoracic aorta (see next image – difficult to identify both penetrating and exit wounds. Peri-aortic hemorrhage.	03/27/2023
123,607	64,712	F64712	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	CARDIOVASCULAR	AORTA	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	12 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Trajectory of BB pellet through aorta, demonstrated by bacteriology swab.	03/27/2023
123,608	64,713	F64713	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	CARDIOVASCULAR	BLOOD	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	13 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Documenting volume of blood (1.25 L) in thorax.	03/27/2023
123,609	64,714	F64714	O’TOOLE	UNIVERSITY OF WYOMING	CANINE	RESPIRATORY	AGENT-FOREIGN BODY	Forensic	32	Acute exsanguination following gunshot	Gunshot trauma	1	1	Forensic; exsanguination; BB pellet; aorta; hemorrhage; lung; ribcage; necropsy	14 – Series of 14 images showing necropsy sequence documenting tract of BB pellet that killed a hound. Malicious shooting by neighbor irritated by trespassing dog. Image of BB pellet. Projectiles found in a forensic necropsy should be photographed (at a minimum), Ideally the round should be kept until legal hearings are complete.	03/27/2023
123,584	64,689	F64689	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	SENSES	EYE	NEOPLASIA, NEOPLASM, TUMOR	32	Undifferentiated carcinoma of conjunctival origin, most likely third eyelid	Neoplasia	1	1	Conjunctiva; eye; globe; carcinoma; third eyelid; keratitis	12 year old Corgi cross with conjunctival mass and chemosis. WSVL#16C10041	03/26/2023
123,585	64,690	F64690	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	FELINE	SKELETAL	BONE-SKULL	TRAUMA	32	Acute fracture, frontal bones. Acute hemorrhage, M. masseter	Acute trauma	1	1	Fracture; skull; frontal bones; squama; subdural hematoma; masseter	Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772	03/26/2023
123,586	64,691	F64691	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	FELINE	SKELETAL	BONE-SKULL	TRAUMA	32	Acute fracture, frontal bones. Acute hemorrhage, M. masseter	Acute trauma	1	1	Fracture; skull; frontal bones; squama; subdural hematoma	Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772	03/26/2023
123,587	64,692	F64692	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	FELINE	SKELETAL	BONE-SKULL	HEMORRHAGE	32	Acute intracranial hemorrhage	Acute trauma	1	1	Fracture; skull; frontal bones; intra-cranial hemorrhage	Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772	03/26/2023
123,588	64,693	F64693	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	FELINE	SKELETAL	BONE-SKULL	HEMORRHAGE	32	Intra-cranial hemorrhage	Acute trauma	1	1	Fracture; skull; frontal bones; intra-cranial hemorrhage	Recently adopted kitten; found dead in litter box. Presumed sequel to a fall. WSVL #17F15772	03/26/2023
123,589	64,694	F64694	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	NERVOUS	CEREBELLUM	DEGENERATION	32	Cerebellar abiotrophy OMIA 000175 (presumed)	Genetic	1	1	Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia	No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505.	03/26/2023
123,590	64,695	F64695	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	NERVOUS	CEREBELLUM	DEGENERATION	32	Cerebellar abiotrophy OMIA 000175 (presumed)	Genetic	1	1	Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia	No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505.	03/26/2023
123,591	64,696	F64696	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	NERVOUS	CEREBELLUM	DEGENERATION	32	Cerebellar abiotrophy OMIA 000175 (presumed)	Genetic	1	1	Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia	No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505.	03/26/2023
123,592	64,697	F64697	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	NERVOUS	CEREBELLUM	DEGENERATION	32	Cerebellar abiotrophy OMIA 000175 (presumed)	Genetic	1	1	Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia	No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505.	03/26/2023
123,593	64,698	F64698	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	NERVOUS	CEREBELLUM	DEGENERATION	32	Cerebellar abiotrophy OMIA 000175 (presumed)	Genetic	1	1	Cerebellar abiotrophy; hypermetria; Angus breed; wide-based stance; head-tremor; ataxia	No grossly identifiable lesions in cerebellum; loss of Purkinje cells histologically; defect identified multiple times in high-dollar progeny from commercial bull breeding establishment in Western USA. Not confirmed genetically as OMIA 000175, but with histological stigmata of same. Windsor PA et al. Review of neurological diseases of ruminant livestock in Australia. VI: postnatal bovine, and ovine and caprine, neurogenetic disorders. Aust Vet J. 2011 89(11):432-438.. WSVL#17B7505.	03/26/2023
123,575	64,680	F64680	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,576	64,681	F64681	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,577	64,682	F64682	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,578	64,683	F64683	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE-SKULL	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate, with foreshortened skull	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,579	64,684	F64684	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE-SKULL	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate, with foreshortened skull and mild inferior prognathia	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,580	64,685	F64685	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE-SKULL	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate, with foreshortened skull and mild inferior prognathia	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,581	64,686	F64686	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,582	64,687	F64687	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	SKELETAL	BONE-HUMERUS	DYSPLASIA, dysplastic	32	Chondrodysplasia, disproportionate	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,583	64,688	F64688	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	CARDIOVASCULAR	HEART	HYPERTROPHY	32	Cardiac hypertrophy and chronic passive congestion	Manganese deficiency	1	1	“Acorn calves”; manganese deficiency; chondrodysplasia; superior brachygnathia; inferior brachygnathia; prognathy; nutritional deficiency; “congenital chondrodystrophy of unknown origin” (CCUO); “congenital joint laxity and deformity” (CJLD)	This series of calves with disproportionate dwarfism had manganese deficiency. The older literature refers to them as “acorn calves.” This is a confusing and misleading term, as there is no direct etiological association with acorns. It derives from cases seen in multiple cattle breeds (beef and dairy) in the oak belt of the Sierra Nevada foothills in the US during the 1930s (Hart et al., 1947, “Acorn calves.” A nonhereditary congenital deformity due to maternal nutritional deficiency. Experiment Station Bulletin 699, College of Agriculture, University of California, Berkley, pp. 1-24). It occurs sporadically in N. America, Australia and New Zealand, South Africa and Europe with an incidence of 5 – 15%. These affected Wyoming calves had hepatic manganese considerably below the reference range. Features originally described are full-term live-birth, disproportionate dwarfism, short skulls, undershot or overshot mandibles (or long and narrow), no common blood line among affected calves, a tendency to be the progeny of heifers, and a history of “poor feed” for extended periods during gestation. Some surviving calves improve if they survive the nursing period. Drought and dry feed are a common factor in the history. Calves may display ordination, inability to stand unaided, kyphosis, a tendency toward, and incoordination with various other neurological signs. Unlike Dexter bulldog calves, affected acorn calves are born alive and full term. The cases shown here, from a large episode, were from one herd where dams were on suboptimal feed in a Nebraska feedlot. Ref: White PJ, Windsor PA:. Congenital chondrodystrophy of unknown origin in beef herds. Vet J. 2012. 193(2):336-43.	03/25/2023
123,569	64,674	F64674	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	CANINE	RESPIRATORY	LARYNX	FOREIGN BODY	32	Foreign body aspiration (food)	Aspiration of food	1	1	Foreign body; aspiration; laryngospasm	This was a nursing puppy transitioning to solid feed. Feed particles were predominantly in larynx, rather than lung. I assume the puppy died due to a combination of laryngospasm and partial obstruction by feed particles of larynx	03/24/2023
123,570	64,675	F64675	O’TOOLE	WYOMING STATE VETERINARY LABORATORY	BOVINE	GASTROINTESTINAL	CECUM	METABOLIC DISEASE	32	Multifocal superficial ulcerative typhlitis secondary to uremia	Uremia (urolithiasis) resulting in a ruptured bladder and uroperitoneum	1	1	Uremia; cecum; typhlitis; vasculitis; urolithiasis	Cattle and horses are prone to ulcerative or hemorrhagic typhlitis-colitis as a result of uremia. By contrast, oral, lingual and gastric mucosa are the main tissues developing ulcers or hemorrhagic lesions in uremic dogs, associated with vascular lesions. WSVL#16B6800	03/24/2023
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